The Sun Keeps Rising: Why Seniors Can't Skip UV Protection

Senior Sun Protection

Congratulations. You’ve survived life’s slings and arrows, and made it to your senior years. It took some brains. It took common sense. Now is not the time to abandon those assets.

Many older people seem to feel that after navigating past decades of life’s pitfalls, they can cast caution to the winds – especially when it comes to sun exposure. The thinking may go like this: “I’ve never had skin cancer. It takes decades for skin cancer to develop, so I’m never going to get it. I’m moving to Arizona and reveling in the sun.”

THE DOWNSIDE OF LONGER LIFE

The first flaw in that thinking is that none of us know how long we will live; Mickey Mantle, who died of cancer at age 63, famously said, “If I knew I was going to live this long, I’d have taken better care of myself.” We need to keep taking care of ourselves to extend our lives and stay strong and healthy as long as possible.

The average lifespan in the industrial world has been rising steeply. By 2020, 25% of the US workforce will be composed of older workers1— sometimes called the Silver Tsunami—and epidemiological, biological and molecular data all point to skin cancer as predominantly a disease of the elderly. Between 40% and 50% of Americans who live to age 65 will have skin cancer at least once.2 Caucasian men over age 65 have had a 5.1% annual increase in melanoma incidence since 1975— the highest annual increase of any gender or age group.3 It has also been reported that more than half of skin cancer-related deaths occur in persons more than 65 years old.4 The longer people live, the more likely they are to develop skin cancer, and the greater their chances of dying from it.

There are many reasons for this. First, most skin cancers result from sun damage over the course of our lives, and seniors have lived longer; they have had the most sun exposure and sustained the most damage from ultraviolet (UV) light. Both sunburns and suntans damage our skin’s DNA, breaking down the skin’s tissues so that it ages before its time, and producing genetic defects that can lead to skin cancer. Suffering just five sunburns over your lifetime more than doubles your chances of developing melanoma, and each successive tan or sunburn raises the risks further.5 We never know exactly how much damage will trigger a skin cancer, but studies show that one bad burn in older age may be the straw that broke the camel’s back.5

DIMINISHED DEFENSES

aging back

Making matters worse, as the damage mounts, our ability to stave it off keeps diminishing. As we age, our skin undergoes changes that weaken our defenses against skin disease: reduced immune systems, poorer healing capacity, thinner skin, and damage from bodily assaults from smoking to pollution.6,7 These changes all contribute to accelerated skin aging and increase our risk for skin cancer.8

INTRINSIC VS. EXTRINSIC AGING

Two types of skin aging exist: intrinsic, or normal chronological aging, which occurs in all individuals, and extrinsic aging, caused by external factors such as ultraviolet (UV) light exposure (both sunlight and tanning beds), industrial chemicals, human immunodeficiency virus and environmental pollutants.8 Both play a part in skin cancer.

Intrinsic Aging: In our advanced years, our skin loses fat and water content and becomes thinner, allowing UV light to penetrate more deeply. Compounding the problem, the body’s natural ability to repair damaged DNA diminishes, increasing the likelihood of abnormal cell growth that can cause mutations leading to skin cancer. The overall natural decline in our immune systems not only may allow prior DNA damage to progress to cancer, but leaves us more susceptible to cancers from future DNA damage. Many diseases and conditions related to aging contribute to this immune decline. Atherosclerosis, diabetes mellitus, and congestive heart failure, for example, are known to impede blood flow and decrease immune responses, reducing the skin’s ability to heal.

Extrinsic Aging: If all that’s not bad enough, we regularly expose our skin to agents that further weaken our defenses. Above all, many older individuals vastly increase their UV exposure, moving to sunnier climes and engaging in more outdoor activities like golf, fishing, and tennis. Since UV light itself suppresses the immune system, this exacerbates our natural immune decline and facilitates the development of skin cancer. UV light also breaks down elastic tissue (elastin) in the skin over time, leading to wrinkles, sagging, discoloration, and blotchiness.

The skin is especially susceptible to sun damage since it covers the entire surface of the body; it is the first organ to come in direct contact with UV rays. We once thought that most sun damage occurred before age 18, and that this early damage triggered most of the genetic changes that later led to skin precancers and cancers. That left some older people thinking, “The damage is done, and there’s nothing I can do about it.” However, subsequent research showed that we continue to have substantial UV exposure as long as we live; the majority of exposure occurs after age 40.9 This later exposure is often what kicks on skin cancers, so sun protection remains vital throughout our lives.

PREVENTION: NOT THAT TOUGH

Senior Sun ProtectionSince we know that UV exposure is its primary cause, skin cancer is almost entirely preventable. Fortunately for older people, prevention is not that big a burden. It just takes some consistent precaution.10 It’s pretty much a three-pronged program: 1) stay out of tanning beds, 2) use effective sun protection, and 3) check your skin. The first part is exceedingly easy: simply never climb into a tanning bed; more people develop skin cancer because of UV tanning than develop lung cancer because of smoking.11

Proper sun protection starts with timing: The hours between 10 AM and 4 PM are typically the most UV-intense, so plan outside adventures for early morning or late afternoon. When you do go outside, seek shade from the direct sun, and wear sun-safe clothing, including a long-sleeved shirt and long pants made of densely woven materials, a widebrimmed hat, and UV-filtering sunglasses. Use an SPF 15 or higher broad spectrum sunscreen (SPF 30+, water-resistant sunscreen for extended or intense outdoor exposures such as on the golf course), and reapply at least every two hours or immediately after swimming or heavy sweating.

It also helps to be aware of your skin type, since fair-skinned people with light-colored eyes and hair (Types 1 and II) suffer sun damage more easily. Go to SkinCancer.org/quiz to find your skin type.

Sun ProtectionFinally, along with protecting your skin, watch for suspicious growths. The Skin Cancer Foundation recommends head-to-toe self-examination once a month and an annual visit to a dermatologist for a professional total-body exam. This will give you the best chance of discovering skin cancer at an early, easily treatable stage. Be alert to any growth with an irregular border, multiple colors, and increased size or any other notable change. Persistent pain, irritation, itching, bleeding or crusting at any skin site should also be brought to your dermatologist’s attention, as well as any new lesion appearing after age 40.

The dermatologist can also partially repair some of your lifelong sun damage, using techniques such as lasers and photodynamic therapy, dermabrasion, and topical medications like retinoids, helping to rejuvenate your skin while also removing precancerous lesions, thereby reducing your risk of skin cancer.

CONCLUSIONS

Senior Sun ProtectionYou’ve probably heard more than one older person say something like, “When I was young, no one used sunscreen. It’s too late to change the past, so if I get skin cancer, I get it.” You now know that’s not true; it’s never too late to reduce your skin cancer risk.

Follow our advice, and you will vastly improve your chances of avoiding skin cancer or catching it when it is easily treatable. I also want to emphasize the importance of overall health. Well-balanced nutrition, good sleep, and UV protection, for example, help keep your immune system strong so that you can better fight off skin diseases. Also, dangerous habits like smoking, excessive alcohol consumption, and drug dependency all contribute to dermatological issues. The bad effects of bad habits add up, increasing your chance of any and all diseases, including skin cancers. So take care of yourself.

Published on August 14, 2015

References

  1. Hayutin A, Beals M, Borges E. The aging US workforce. A chartbook of demographic shifts. Introduction. Stanford Center on Longevity 2013. http://longevity3.stanford.edu/wp-content/uploads/2014/01/The_Aging_U.S.-Workforce.pdf. Accessed March 12, 2015.
  2. Sun Protection. Cancer Trends Progress Report –2009/2010 Update. National Cancer Institute. http://progressreport.cancer.gov/sites/default/files/archive/report2009.pdf Accessed November 1, 2010.
  3. National Cancer Institute. A snapshot of melanoma. http://www.cancer.gov/aboutnci/servingpeople/snapshots/melanoma.pdf. updated Oct 2011; accessed Aug 27, 2012.
  4. Syrigos KN, Tzannou I, Katirtzoglou N, Georgiou E. Skin cancer in the elderly. In Vivo 2005; 19(3):643-52.
  5. Pfahlberg A, Kolmel KF, Gefeller O. Timing of excessive ultraviolet radiation and melanoma: epidemiology does not support the existence of a critical period of high susceptibility to solar ultraviolet radiation-induced melanoma. Brit J Dermatol March 2001; 144:3:471.
  6. Merimsky O, Inbar M. Cigarette smoking and skin cancer. Clin Dermatol 1998;16(5):585-8.
  7. Boyd AS, Stasko T, Lloyd E, et al. Cigarette smoking– associated elastotic changes in the skin. J Am Acad Dermatol 1999; 41:23-6.
  8. Norman R. 100 Questions & Answers about Aging Skin. Jones and Bartlett Publishers LLC 2010; Sudbury, MA.
  9. Godar DE, Urbach F, Gasparro FP, Van der Leun JC. UV doses of young adults. Photochem Photobiol 2003; 77(4):453-457.
  10. Norman R. Preventive Dermatology. London, UK: Springer; 2010.
  11. Wehner M, Chren M-M, Nameth D, et al. Internationalprevalence of indoor tanning: a systematic reviewand meta-analysis. JAMA Dermatol 2014; 150(4):390-400. Doi: 10.1001/jamadermatol.2013.6896.